Number of items: 22.
2022
Ismail, N. F. B., Foth, M., Yousef, A. R. E., Cui, N., Leach, J. D.G., Jamieson, T., Karim, S. A., Salmond, J. M., Morton, J. P. and Iwata, T.
(2022)
Loss of Cxcr2 in myeloid cells promotes tumour progression and T cell infiltration in invasive bladder cancer.
Bladder Cancer, 8(3),
pp. 277-290.
(doi: 10.3233/BLC-211645)
2020
Pritchard, J. J.G., Hamilton, G., Hurst, C. D., Fraser, S., Orange, C., Knowles, M. A., Jones, R. J. , Leung, H. Y. and Iwata, T.
(2020)
Monitoring of urothelial cancer disease status after treatment by digital droplet PCR liquid biopsy assays.
Urologic Oncology: Seminars and Original Investigations, 38(9),
731.e1-737.e10.
(doi: 10.1016/j.urolonc.2020.05.012)
(PMID:32532529)
2018
Foth, M., Ismail, N. F. B., Kung, J. S. C., Tomlinson, D., Knowles, M. A., Eriksson, P., Sjödahl, G., Salmond, J. M., Sansom, O. J. and Iwata, T.
(2018)
FGFR3 mutation increases bladder tumorigenesis by suppressing acute inflammation.
Journal of Pathology, 246(3),
pp. 331-343.
(doi: 10.1002/path.5143)
(PMID:30043421)
(PMCID:PMC6334176)
2016
Rehbini, O., Mohan, G., Sindi, A., Garza Manero, S., Gurden, R., Iwata, T. and West, K.
(2016)
Knockdown High Mobility Nucleosomal Binding Proteins 2 (HMGN2) Alter the Histone Modification H4K4me3 and H3K27me3 and Regulates Stem Cells Pluripotency.
9th Saudi Student Conference, Birmingham, UK, 13-14 Feb 2016.
2015
Rehbini, O., Sindi, A., Mohan, G., Garza Manero, S., Iwata, T. and West, K. L.
(2015)
The Hmgn Family of Chromatin Binding Proteins Regulates Stem Cells Pluripotency and Neuronal Differentiation.
Wellcome Trust-Waddington Symposium: Epigenetics in Dialogue with THE GENOME, Edinburgh, UK, 01-05 Jun 2015.
2014
Foth, M. et al.
(2014)
Fibroblast growth factor receptor 3 activation plays a causative role in urothelial cancer pathogenesis in cooperation with Pten loss in mice.
Journal of Pathology, 233(2),
pp. 148-158.
(doi: 10.1002/path.4334)
2012
Mohan, G., Rehbini, O., Iwata, T. and West, K.
(2012)
The Hmgn Family of Chromatin Binding Proteins Regulate Stem Cell Pluripotency and Neuronal Differentiation.
Biochemical Society Annual Symposium: Epigenetic Mechanisms in Development and Disease, Leeds, UK, 11-13 Dec 2012.
Ahmad, I. , Iwata, T. and Leung, H.Y.
(2012)
Mechanisms of FGFR-mediated carcinogenesis.
Biochimica et Biophysica Acta: Molecular Cell Research, 1823(4),
pp. 850-860.
(doi: 10.1016/j.bbamcr.2012.01.004)
2011
Ahmad, I. , Singh, L.B., Foth, M., Morris, C.-A., Taketo, M.M., Wu, X.-R., Leung, H.Y. , Sansom, O.J. and Iwata, T.
(2011)
K-Ras and β-catenin mutations cooperate with Fgfr3 mutations in mice to promote tumorigenesis in the skin and lung, but not in the bladder.
Disease Models and Mechanisms, 4(4),
pp. 548-555.
(doi: 10.1242/dmm.006874)
Moldrich, R.X. et al.
(2011)
Fgfr3 regulates development of the caudal telencephalon.
Developmental Dynamics, 240(6),
pp. 1586-1599.
(doi: 10.1002/dvdy.22636)
(PMID:21491541)
2010
Pellicano, F., Thomson, R.E., Inman, G.J. and Iwata, T.
(2010)
Regulation of cell proliferation and apoptosis in neuroblastoma cells by ccp1, a FGF2 downstream gene.
BMC Cancer, 10(1),
657.
(doi: 10.1186/1471-2407-10-657)
(PMID:21118521)
(PMCID:PMC3001724)
2009
Iwata, T. and Hevner, R.
(2009)
Fibroblast growth factor signaling in development of the cerebral cortex.
Development Growth and Differentiation, 51(3),
pp. 299-323.
Thomson, R., Kind, P.C., Graham, N.A., Etherson, M.L., Kennedy, J., Fernandes, A.C., Marques, C.S., Hevner, R.F. and Iwata, T.
(2009)
Fgf receptor 3 activation promotes selective growth and expansion of occipitotemporal cortex.
Neural Development, 4(4),
(doi: 10.1186/1749-8104-4-4)
(PMID:19192266)
(PMCID:PMC2661882)
2007
Thomson, R. E., Pellicano, F. and Iwata, T.
(2007)
Fibroblast growth factor receptor 3 kinase domain mutation increases cortical progenitor proliferation via mitogen-activated protein kinase activation.
Journal of Neurochemistry, 100(6),
pp. 1565-1578.
(doi: 10.1111/j.1471-4159.2006.04285.x)
(PMID:17181553)
2006
Ambrosino, C., Iwata, T., Scafoglio, C., Mallardo, M., Klein, R. and Nebreda, A.
(2006)
TEF-1 and C/EBPbeta are major p38alpha MAPK-regulated transcription factors in proliferating cardiomyocytes.
Biochemical Journal, 396(1),
pp. 163-172.
Pellicano, F., Inglis-Broadgate, S. L., Pante, G., Ansorge, W. and Iwata, T.
(2006)
Expression of coiled-coil protein 1, a novel gene downstream of FGF2, in the developing brain.
Gene Expression Patterns, 6(3),
pp. 285-293.
(doi: 10.1016/j.modgep.2005.07.004)
2005
Inglis-Broadgate, S. L., Thomson, R. E., Pellicano, F., Tartaglia, M. A., Pontikis, C. C., Cooper, J. D. and Iwata, T.
(2005)
FGFR3 regulates brain size by controlling progenitor cell proliferation and apoptosis during embryonic development.
Developmental Biology, 279(1),
pp. 73-85.
(doi: 10.1016/j.ydbio.2004.11.035)
Pante, G., Thompson, J., Lamballe, F., Iwata, T., Ferby, I., Barr, F., Davies, A., Maina, F. and Klein, R.
(2005)
Mitogen-inducible gene 6 is an endogenous inhibitor of HGF/Met-induced cell migration and neurite growth.
Journal of Cell Biology, 171,
pp. 337-348.
2004
Cho, J., Guo, C., Torello, M., Lunstrum, G., Iwata, T., Deng, C. and Horton, W.
(2004)
Defective lysosomal targeting of activated fibroblast growth factor receptor 3 in achondroplasia.
Proc Natl Acad Sci U S A, 101(2),
pp. 609-614.
2001
Iwata, T., Li, C., Deng, C. and Francomano, C.
(2001)
Highly activated Fgfr3 with the K644M mutation causes prolonged survival in severe dwarf mice.
Human Molecular Genetics, 10(12),
pp. 1255-1264.
2000
Iwata, T. , Chen, L., Li, C., Ovchinnikov, D.A., Behringer, R.R., Francomano, C.A. and Deng, C.X.
(2000)
A neonatal lethal mutation in FGFR3 uncouples proliferation and differentiation of growth plate chondrocytes in embryos.
Human Molecular Genetics, 9(11),
pp. 1603-13.
1999
Li, C., Chen, L., Iwata, T. , Kitagawa, M., Fu, X.Y. and Deng, C.X.
(1999)
A Lys644Glu substitution in fibroblast growth factor receptor 3 (FGFR3) causes dwarfism in mice by activation of STATs and ink4 cell cycle inhibitors.
Human Molecular Genetics, 8(1),
pp. 35-44.
This list was generated on Thu May 16 19:11:19 2024 BST.