Dr Wai Kwong Lee

Research Manager (Cardiovascular & Metabolic Health Professional Services)

telephone: 01413308275
email: Wai.Lee@glasgow.ac.uk

School of Cardiovascular & Metabolic Health, BHF Glasgow Cardiovascular Reserach Centre, 126 University Place, University of Glasgow, Glasgow, G12 8TA

Import to contacts

https://orcid.org/0000-0001-8560-8063

Publications

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Jump to: 2020 | 2017 | 2014 | 2013 | 2011 | 2010 | 2008 | 2005 | 1996

Number of items: 14.

2020

Steinthorsdottir, V. et al. (2020) Genetic predisposition to hypertension is associated with preeclampsia in European and Central Asian women. Nature Communications, 11, 5976. (doi: 10.1038/s41467-020-19733-6) (PMID:33239696) (PMCID:PMC7688949)

2017

McGinnis, R. et al. (2017) Variants in the fetal genome near FLT1 are associated with risk of preeclampsia. Nature Genetics, 49(8), pp. 1255-1260. (doi: 10.1038/ng.3895) (PMID:28628106)

2014

Arking, D. E. et al. (2014) Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization. Nature Genetics, 46(8), pp. 826-836. (doi: 10.1038/ng.3014) (PMID:24952745) (PMCID:PMC4124521)

2013

Alvarez-Madrazo, S. et al. (2013) Common polymorphisms in the CYP11B1 and CYP11B2 genes: evidence for a digenic influence on hypertension. Hypertension, 61(1), pp. 232-239. (doi: 10.1161/HYPERTENSIONAHA.112.200741)

2011

Johnson, T. et al. (2011) Blood pressure loci identified with a gene-centric array. American Journal of Human Genetics, 89(6), pp. 688-700. (doi: 10.1016/j.ajhg.2011.10.013)

Grassi, G. et al. (2011) Association between ADRA1A gene and the metabolic syndrome: candidate genes and functional counterpart in the PAMELA population. Journal of Hypertension, 29(6), pp. 1121-1127777777777. (doi: 10.1097/HJH.0b013e328346d72c)

Menni, C. et al. (2011) Evaluation of How Gene-Job Strain Interaction Affects Blood Pressure in the PAMELA Study. Psychosomatic Medicine, 73(4), pp. 304-309. (doi: 10.1097/PSY.0b013e318212e0be)

2010

Kane, N. M. et al. (2010) Lentivirus-mediated reprogramming of somatic cells in the absence of transgenic transcription factors. Molecular Therapy, 18(12), pp. 2139-2145. (doi: 10.1038/mt.2010.231)

Padmanabhan, S. et al. (2010) Genome-wide association study of blood pressure extremes identifies variant near UMOD associated with hypertension. PLoS Genetics, 6(10), e1001177. (doi: 10.1371/journal.pgen.1001177)

Padmanabhan, S. et al. (2010) The effects of sex and method of blood pressure measurement on genetic associations with blood pressure in the PAMELA study. Journal of Hypertension, 28(3), pp. 465-477. (doi: 10.1097/HJH.0b013e32833594d7)

2008

Delles, C. et al. (2008) Glutathione S-transferase variants and hypertension. Journal of Hypertension, 26(7), pp. 1343-1352. (doi: 10.1097/HJH.0b013e3282fe1d67)

Padmanabhan, S. , Menni, C., Lee, W., Brambilla, P., Laing, S., Sega, R., Cesana, G., Mancia, G. and Dominiczak, A. (2008) Cardiovascular gene centric association study of metabolic syndrome and ambulatory blood pressure in the PAMELA study. Journal of Hypertension, 26, S18-S18.

2005

Dominiczak, A. F., Graham, D. , McBride, M. W. , Brain, N. J.R., Lee, W. K. , Charchar, F. J., Tomaszewski, M., Delles, C. and Hamilton, C. A. (2005) Corcoran Lecture. Cardiovascular genomics and oxidative stress. Hypertension, 45(4), pp. 636-642. (doi: 10.1161/01.HYP.0000154253.53134.09)

1996

Dominiczak, A. F. , Devlin, A. M., Lee, W. K. , Anderson, N. H., Bohr, D. F. and Reid, J. L. (1996) Vascular smooth muscle polyploidy and cardiac hypertrophy in genetic hypertension. Hypertension, 27(3), pp. 752-759. (doi: 10.1161/01.HYP.27.3.752) (PMID:8613236)

This list was generated on Sun Dec 22 12:29:59 2024 GMT.

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