Ms Annette Lake
- CL3 Coordinator (Virology)
telephone:
01413305082
email:
Annette.Lake@glasgow.ac.uk
Centre for Virus Research, Room 204, Sir Michael Stoker Building, 464 Bearsden Road, Glasgow, G61 1QH
Research interests
Member of the Jarrett Group.
Publications
2022
Berndt, S. I. et al. (2022) Distinct germline genetic susceptibility profiles identified for common non-Hodgkin lymphoma subtypes. Leukemia, 36(12), pp. 2835-2844. (doi: 10.1038/s41375-022-01711-0) (PMID:36273105)
2021
Niemi, M. E. K. et al. (2021) Mapping the human genetic architecture of COVID-19. Nature, 600(7889), pp. 472-477. (doi: 10.1038/s41586-021-03767-x) (PMID:34237774) (PMCID:PMC8674144)
Rinaldi, C., Corrigan, D. K., Dennany, L., Jarrett, R. F. , Lake, A. and Baker, M. J. (2021) Development of an electrochemical CCL17/TARC biosensor toward rapid triage and monitoring of classic Hodgkin lymphoma. ACS Sensors, 6(9), pp. 3262-3272. (doi: 10.1021/acssensors.1c00972) (PMID:34478275)
2020
Liu, Z. et al. (2020) Evaluation of the antibody response to the EBV proteome in EBV-associated classic Hodgkin lymphoma. International Journal of Cancer, 147(3), pp. 608-618. (doi: 10.1002/ijc.32741) (PMID:31618442)
2017
Sud, A. et al. (2017) Genome-wide association study of classical Hodgkin lymphoma identifies key regulators of disease susceptibility. Nature Communications, 8, 1892. (doi: 10.1038/s41467-017-00320-1) (PMID:29196614) (PMCID:PMC5711884)
2015
Johnson, P. C.D. et al. (2015) Modeling HLA associations with EBV-positive and -negative Hodgkin lymphoma suggests distinct mechanisms in disease pathogenesis. International Journal of Cancer, 137(5), pp. 1066-1075. (doi: 10.1002/ijc.29467) (PMID:25648508) (PMCID:PMC4737225)
2014
Bell, A. J., Gallagher, A., Mottram, T., Lake, A., Kane, E. V., Lightfoot, T., Roman, E. and Jarrett, R. F. (2014) Germ-line transmitted, chromosomally integrated hhv-6 and classical Hodgkin lymphoma. PLoS ONE, 9(11), e112642. (doi: 10.1371/journal.pone.0112642) (PMID:25384040) (PMCID:PMC4226568)
2013
Frampton, M. et al. (2013) Variation at 3p24.1 and 6q23.3 influences the risk of Hodgkin’s lymphoma. Nature Communications, 4, (doi: 10.1038/ncomms3549)
2012
Urayama, K. Y. et al. (2012) Genome-wide association study of classical Hodgkin lymphoma and Epstein-Barr virus status-defined subgroups. Journal of the National Cancer Institute, 104(3), pp. 240-253. (doi: 10.1093/jnci/djr516)
2010
Enciso-Mora, V. et al. (2010) A genome-wide association study of Hodgkin's lymphoma identifies new susceptibility loci at 2p16.1 (REL), 8q24.21 and 10p14 (GATA3). Nature Genetics, 42(12), pp. 1126-1130. (doi: 10.1038/ng.696)
Hjalgrim, H. et al. (2010) HLA-A alleles and infectious mononucleosis suggest a critical role for cytotoxic T-cell response in EBV-related Hodgkin lymphoma. Proceedings of the National Academy of Sciences of the United States of America, 107(14), pp. 6400-6405. (doi: 10.1073/pnas.0915054107)
2009
Lake, A. et al. (2009) Mutations of NFKBIA, encoding I kappa B alpha, are a recurrent finding in classical Hodgkin lymphoma but are not a unifying feature of non-EBV-associated cases. International Journal of Cancer, 125(6), pp. 1334-1342. (doi: 10.1002/ijc.24502)
Articles
Berndt, S. I. et al. (2022) Distinct germline genetic susceptibility profiles identified for common non-Hodgkin lymphoma subtypes. Leukemia, 36(12), pp. 2835-2844. (doi: 10.1038/s41375-022-01711-0) (PMID:36273105)
Niemi, M. E. K. et al. (2021) Mapping the human genetic architecture of COVID-19. Nature, 600(7889), pp. 472-477. (doi: 10.1038/s41586-021-03767-x) (PMID:34237774) (PMCID:PMC8674144)
Rinaldi, C., Corrigan, D. K., Dennany, L., Jarrett, R. F. , Lake, A. and Baker, M. J. (2021) Development of an electrochemical CCL17/TARC biosensor toward rapid triage and monitoring of classic Hodgkin lymphoma. ACS Sensors, 6(9), pp. 3262-3272. (doi: 10.1021/acssensors.1c00972) (PMID:34478275)
Liu, Z. et al. (2020) Evaluation of the antibody response to the EBV proteome in EBV-associated classic Hodgkin lymphoma. International Journal of Cancer, 147(3), pp. 608-618. (doi: 10.1002/ijc.32741) (PMID:31618442)
Sud, A. et al. (2017) Genome-wide association study of classical Hodgkin lymphoma identifies key regulators of disease susceptibility. Nature Communications, 8, 1892. (doi: 10.1038/s41467-017-00320-1) (PMID:29196614) (PMCID:PMC5711884)
Johnson, P. C.D. et al. (2015) Modeling HLA associations with EBV-positive and -negative Hodgkin lymphoma suggests distinct mechanisms in disease pathogenesis. International Journal of Cancer, 137(5), pp. 1066-1075. (doi: 10.1002/ijc.29467) (PMID:25648508) (PMCID:PMC4737225)
Bell, A. J., Gallagher, A., Mottram, T., Lake, A., Kane, E. V., Lightfoot, T., Roman, E. and Jarrett, R. F. (2014) Germ-line transmitted, chromosomally integrated hhv-6 and classical Hodgkin lymphoma. PLoS ONE, 9(11), e112642. (doi: 10.1371/journal.pone.0112642) (PMID:25384040) (PMCID:PMC4226568)
Frampton, M. et al. (2013) Variation at 3p24.1 and 6q23.3 influences the risk of Hodgkin’s lymphoma. Nature Communications, 4, (doi: 10.1038/ncomms3549)
Urayama, K. Y. et al. (2012) Genome-wide association study of classical Hodgkin lymphoma and Epstein-Barr virus status-defined subgroups. Journal of the National Cancer Institute, 104(3), pp. 240-253. (doi: 10.1093/jnci/djr516)
Enciso-Mora, V. et al. (2010) A genome-wide association study of Hodgkin's lymphoma identifies new susceptibility loci at 2p16.1 (REL), 8q24.21 and 10p14 (GATA3). Nature Genetics, 42(12), pp. 1126-1130. (doi: 10.1038/ng.696)
Hjalgrim, H. et al. (2010) HLA-A alleles and infectious mononucleosis suggest a critical role for cytotoxic T-cell response in EBV-related Hodgkin lymphoma. Proceedings of the National Academy of Sciences of the United States of America, 107(14), pp. 6400-6405. (doi: 10.1073/pnas.0915054107)
Lake, A. et al. (2009) Mutations of NFKBIA, encoding I kappa B alpha, are a recurrent finding in classical Hodgkin lymphoma but are not a unifying feature of non-EBV-associated cases. International Journal of Cancer, 125(6), pp. 1334-1342. (doi: 10.1002/ijc.24502)
